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Attributs	Valeurs
type	work Issue
Is Part Of	http://hub.abes.fr/oup/periodical/cardiovascres/2006/volume_69
has manifestation of work	http://hub.abes.fr/oup/periodical/cardiovascres/2006/volume_69/issue_3/m/web http://hub.abes.fr/oup/periodical/cardiovascres/2006/volume_69/issue_3/m/print
Date Copyrighted	2005 2006
Rights	Copyright © 2005, European Society of Cardiology Copyright © 2006, European Society of Cardiology
issue	3
is Is Part Of of	Structure and function of matrix metalloproteinases and TIMPs Instructions to Authors Influence of matrix metalloproteinase genotype on cardiovascular disease susceptibility and outcome Integrins, membrane-type matrix metalloproteinases and ADAMs: Potential implications for cardiac remodeling Chemokines induce matrix metalloproteinase-2 through activation of epidermal growth factor receptor in arterial smooth muscle cells Pathways of matrix metalloproteinase induction in heart failure: Bioactive molecules and transcriptional regulation Myocardial proteases and matrix remodeling in inflammatory heart disease Cardiac mast cell regulation of matrix metalloproteinase-related ventricular remodeling in chronic pressure or volume overload Editorial Board Relevance of matrix metalloproteinases and their inhibitors after myocardial infarction: A temporal and spatial window Spotlight Issue on Role of Nutrition in Mechanisms of Cardiovascular Disease Pravastatin increases survival and suppresses an increase in myocardial matrix metalloproteinase activity in a rat model of heart failure Degraded collagen induces calpain-mediated apoptosis and destruction of the X-chromosome-linked inhibitor of apoptosis (xIAP) in human vascular smooth muscle cells Matrix metalloproteinases regulate migration, proliferation, and death of vascular smooth muscle cells by degrading matrix and non-matrix substrates What has been learned about the cardiovascular effects of matrix metalloproteinases from mouse models? Atherosclerosis and proteinase activation Vascular remodeling and protease inhibition-bench to bedside From tadpole tails to transgenic mice: Metalloproteinases have brought about a metamorphosis in our understanding of cardiovascular disease Short-term treatment of spontaneously hypertensive rats with liver growth factor reduces carotid artery fibrosis, improves vascular function, and lowers blood pressure Cardiac transgenic matrix metalloproteinase-2 expression directly induces impaired contractility Direct effects of leptin on size and extracellular matrix components of human pediatric ventricular myocytes The importance of estimating the therapeutic index in the development of matrix metalloproteinase inhibitors Inflammation and postinfarct remodeling: Overexpression of IκB prevents ventricular dilation via increasing TIMP levels Fibulin-2 is present in murine vascular lesions and is important for smooth muscle cell migration Matrix metalloproteinase-2 and -9 are induced differently by doxorubicin in H9c2 cells: The role of MAP kinases and NAD(P)H oxidase

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