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Attributs	Valeurs
type	work Issue
Is Part Of	http://hub.abes.fr/oup/periodical/ndt/1996/volume_11
has manifestation of work	http://hub.abes.fr/oup/periodical/ndt/1996/volume_11/issue_supp6/m/web http://hub.abes.fr/oup/periodical/ndt/1996/volume_11/issue_supp6/m/print
Date Copyrighted	1996
Rights	© 1996 European Renal Association-European Dialysis and Transplant Association
issue	supp6
is Is Part Of of	Detection of two different nonsense mutations in exon 44 of the PKD1 gene in two unrelated Italian families with severe autosomal dominant polycystic kidney disease Outcome of renal replacement therapy in autosomal dominant polycystic kidney disease Murine models of polycystic kidney disease In vitro formation of cysts derived from a rat model of autosomal dominant polycystic kidney disease Molecular genetics of human renal cell tumours Detection of translation terminating mutations in the PKD1 gene Foreword Structural motifs of the PKD1 protein The kidney in tuberous sclerosis: manifestations and molecular genetic mechanisms Autosomal dominant polycystic kidney disease—the patient on renal replacement therapy Anticipation of end-stage renal disease in autosomal dominant polycystic kidney disease Autosomal recessive polycystic kidney disease Rat models of autosomal dominant polycystic kidney disease Analysis of a large family with the second type of autosomal dominant polycystic kidney disease

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