About: Analbuminemia

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Attributs	Valeurs
type	work Article
Is Part Of	http://hub.abes.fr/springer/periodical/109/1983/volume_61/issue_11/w
Subject	Idiopathic hypoalbuminemia Analbuminemia
Title	Analbuminemia
has manifestation of work	http://hub.abes.fr/springer/periodical/109/1983/volume_61/issue_11/B8E7C93BF23350DFE053120B220A7089/m/print http://hub.abes.fr/springer/periodical/109/1983/volume_61/issue_11/B8E7C93BF23350DFE053120B220A7089/m/web
related by	http://hub.abes.fr/springer/periodical/109/1983/volume_61/issue_11/B8E7C93BF23350DFE053120B220A7089/authorship/2 http://hub.abes.fr/springer/periodical/109/1983/volume_61/issue_11/B8E7C93BF23350DFE053120B220A7089/authorship/1
Author	http://hub.abes.fr/springer/periodical/109/1983/volume_61/issue_11/B8E7C93BF23350DFE053120B220A7089/russie http://hub.abes.fr/springer/periodical/109/1983/volume_61/issue_11/B8E7C93BF23350DFE053120B220A7089/weigandk
Abstract	Summary. Analbuminemia (idiopathic hypoalbuminemia) is a rare congenital anomaly of albumin characterised by very low levels of this protein. Only 21 cases have been reported in the literature. The lack of albumin is “compensated” by increased amounts of various circulating plasma proteins. Other not well understood regulatory mechanisms are responsible for the absence of severe clinical consequences. Clinical aspects, genetics and pathophysiology of this disease are discussed.
article type	ReviewPaper Ubersichten
publisher identifier	BF01486843
Date Copyrighted	1983
Rights Holder	Springer-Verlag GmbH & Co KG
is part of this journal	Journal of Molecular Medicine

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