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http://hub.abes.fr/bmj/periodical/annrheumdis/2007/volume_66/issue_2/B35044CA5C3555BDE053120B220AEE5A/authorship/4
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http://hub.abes.fr/bmj/periodical/ard/2012/volume_71/issue_2/B3506C23F2274C7BE053120B220AB6A2/authorship/11
http://hub.abes.fr/oup/periodical/brainj/2004/volume_127/issue_8/101093brainawh193/authorship/1
http://hub.abes.fr/oup/periodical/brheum/2010/volume_49/issue_1/101093rheumatologykep372/authorship/5
http://hub.abes.fr/bmj/periodical/ard/2009/volume_68/issue_2/B35044CA5F4F55BDE053120B220AEE5A/authorship/5
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http://hub.abes.fr/oup/periodical/hmg/2009/volume_18/issue_14/101093hmgddp193/authorship/21
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Association of protein kinase C alpha (PRKCA) gene with multiple sclerosis in a UK population
High resolution linkage and association mapping identifies a novel rheumatoid arthritis susceptibility locus homologous to one linked to two rat models of inflammatory arthritis
Genetics [315-318]
The type 1 diabetes susceptibility gene SUMO4 at IDDM5 is not associated with susceptibility to rheumatoid arthritis or juvenile idiopathic arthritis
Genotype at the sIL-6R A358C polymorphism does not influence response to anti-TNF therapy in patients with rheumatoid arthritis
Re-evaluation of putative rheumatoid arthritis susceptibility genes in the post-genome wide association study era and hypothesis of a key pathway underlying susceptibility
Recent advances in the genetics of RA susceptibility
A single nucleotide polymorphism in exon 1 of cytotoxic T-lymphocyte-associated-4 (CTLA-4) is not associated with rheumatoid arthritis
Association of the tumour necrosis factor-308 variant with differential response to anti-TNF agents in the treatment of rheumatoid arthritis
Investigating the viability of genetic screening/testing for RA susceptibility using combinations of five confirmed risk loci
Identification of AF4/FMR2 family, member 3 (AFF3) as a novel rheumatoid arthritis susceptibility locus and confirmation of two further pan-autoimmune susceptibility genes
Identification of AF4/FMR2 family, member 3 (AFF3) as a novel rheumatoid arthritis susceptibility locus and confirmation of two further pan-autoimmune susceptibility genes
Combined effects of three independent SNPs greatly increase the risk estimate for RA at 6q23
Combined effects of three independent SNPs greatly increase the risk estimate for RA at 6q23
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